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what is the pathophysiology of pneumonia and rheumatic heart disease? Please help me with my homework?

Jun 23, 2007 by pierre! | Posted in Heart Diseases

Who could give me the pathophysiology of pneumonia and Rheumatoid Heart Disease? The diagram and the explanation. Thank you very much.

Check your manual.

Heart Disease Health Center » pathogenesis of rheumatic heart ...

by heart-disease

In encyclopedic on the alert rheumatic fever (ARF) is a late sequela of classifying a Streptococcus (GAS) throat infection. Following a throat infection original, which is often either untreated or partially treated, there are latent while two to three weeks before the first signs of rheumatic fever become sensitive to the outside. A few weeks after the initial symptoms, patients may give with one of the events representative of rheumatic fever alert, including arthritis, carditis, chorea, the subcutaneous nodules and erythema marginatum.(1.2)

Conversion of staff in microbiology from ARF is crucial to the intimacy of the pathophysiology of this disease. GAS is a Gram-positive extracellular pathogen that colonizes the throat or often excoriate. GAS is a living thing that has developed many mechanisms of violence complex became the movement's most daily bacterial pharyngitis, scarlet fever and impetigo. It is lucid GAS strains or serotypes have a predilection remarkably faithful to the well-throat, either in spring or infections fur.In addition, streptococci have been characterized based on proximity to discriminate protein structures M. There are more than eighty types of M protein outstanding gas currently described. The M protein has many functions in bacteria, which include the auspices of master safe return. Specifically, it was demonstrated that the control antibody binding and left deposits derived opsonin, thereby protecting gas against phagocytosis by neutrophils polymorphs.(3)

The prestige of the M protein of gas in the pathogenesis of rheumatic heart disease extends beyond its value in avoiding reaction act exempt. It has been demonstrated that molecular mimicry associated with the accumulation of M protein induced anger reactivity with the horde affected system that results in the murder of cardiac myosin. It has been shown that a short fuse self-reactive antibodies against antigens GAS M protein and heart weight are present in the serum of patients with rheumatic fever.The game of mice and monoclonal antibodies against lethal gases confirmed these reactions cantankerous and identified myosin, tropomyosin, vimentin and heart as autoantigens bored with GAS-reactive protein M....

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